Erosive-ulcerative gastritis with high acidity

Erosive gastritis is an inflammatory disease. It affects the gastric mucosa, causing the formation of erosions of various sizes on it. These formations do not penetrate deep into the walls of the stomach and do not leave marks after healing.

It is the depth of penetration into the stomach wall that distinguishes erosive gastritis from an ulcer. The latter disease can affect not only the inner surface, but also deeper tissues and even the muscle layer.

Erosive gastritis is dangerous due to complications. Statistics show that a fifth of gastric bleeding is associated with erosions on the mucous membrane. Therefore, in modern gastroenterology, this form of the disease is increasingly referred to as “hemorrhagic gastropathy or gastritis” and “papular gastropathy.”

Erosive gastritis is a common pathology. A quarter of examinations for heartburn, abdominal pain, and nausea end with this diagnosis. The disease affects both men and women. The disease can develop at any age. True, young children are diagnosed much less often.

Studies show that in recent years, cases of diagnosing erosive gastritis have become more frequent. According to some studies, the incidence of diseases has increased tenfold. Scientists attribute this to an increase in the amount of stress, disruption of diet and quality of nutrition. This may also be due to improved diagnostic methods. When carrying out preventive examinations, the disease can be identified at the initial stage, even in the absence of complaints.

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Causes of erosive gastritis

In the vast majority of cases, the development of the disease is caused by Helicobacter pylori. However, doctors tend to believe that the mere presence of bacteria in the stomach is not enough. For the disease to appear, a combination of several factors is necessary.

The bacterium Helicobacter pylori was isolated in 1981. It is resistant to acidic conditions and is capable of secreting enzymes that weaken the protective environment of the stomach walls. By multiplying, this microorganism causes local inflammation and the formation of erosions. Helicobacter pylori is quite resistant to antibacterial drugs, which makes treatment difficult.

Research shows that more than 70% of the world's population is infected with Helicobacter pylori. At the moment, this is the most common infection. However, for various reasons, in most cases the presence of bacteria in the body does not cause the development of the disease and infection proceeds unnoticed.

In addition to the presence of pathogenic microorganisms in the stomach, the development of erosive gastritis can also be provoked by other reasons:

  • severe stress
  • chronic mental stress
  • regular alcohol intake
  • eating disorder with long periods of fasting
  • eating spicy and salty foods
  • smoking in the morning before breakfast
  • taking certain medications
  • liver and pancreas diseases
  • chronic gastrointestinal diseases
  • diabetes
  • harmful working conditions
  • reflux of the contents of the duodenum into the stomach

The combination of infection by pathogenic microorganisms with provoking factors reduces the protective function of the body. There is a release of bile, increased production of pepsin and acid. This significantly impairs the ability of the epithelium to regenerate and produce protective mucus and leads to the appearance of defects in the gastric mucosa and the formation of erosive gastritis.

Authorized Products

Nutrition for erosive gastritis with high acidity includes inclusion in the diet:

  • Weak vegetable broths and first courses based on it, to which carefully pureed cereals (white rice, semolina, oatmeal, buckwheat), vermicelli/thin noodles, vegetables are added and seasoned with butter or an egg-milk mixture. It is allowed to add dill and pureed meat in small quantities.
  • Lean meats (chicken, beef, turkey, lamb) in the form of boiled/steamed cutlets and baked dishes. The meat must be pre-boiled and then subjected to further cooking.
  • Low-fat river/sea fish, with the skin removed and steamed/baked in pieces or chopped in the form of cutlets, meatballs, zraz.
  • Porridges boiled in water to a semi-viscous state (white rice, buckwheat, oatmeal), vermicelli/thin noodles in the form of baked krupenik with cottage cheese and puddings.
  • Various boiled vegetables (new peas, potatoes, beets, cauliflower, carrots), prepared in pureed form (puree) with the addition of butter.
  • Dried/day-old white bread, cookies and biscuits.
  • Non-acidic fermented milk and dairy products (fresh cottage cheese, yogurt, kefir), sour cream only in ready-made dishes. It is allowed to eat dishes made from cottage cheese (puddings, cheesecakes, dumplings), occasionally mild cheese in grated form, and eggs in the form of a steam omelet.
  • Non-acidic desserts from pureed berries/fruits, baked/stewed or in the form of jelly, puree, jelly, compotes. It is allowed to eat non-sour jam, meringues, honey, marshmallows, and marshmallows.
  • Rosehip decoction, fruit juices, weak tea/coffee with milk.

Table of permitted products

Proteins, gFats, gCarbohydrates, gCalories, kcal

Vegetables and greens

zucchini0,60,34,624
cauliflower2,50,35,430
potato2,00,418,180
carrot1,30,16,932
beet1,50,18,840
pumpkin1,30,37,728

Fruits

apricots0,90,110,841
bananas1,50,221,895
nectarine0,90,211,848
peaches0,90,111,346
apples0,40,49,847

Berries

strawberry0,80,47,541
raspberries0,80,58,346

Cereals and porridges

buckwheat (kernel)12,63,362,1313
semolina10,31,073,3328
cereals11,97,269,3366
white rice6,70,778,9344

Flour and pasta

noodles12,03,760,1322

Bakery products

white bread crackers11,21,472,2331

Confectionery

jam0,30,263,0263
jelly2,70,017,979
marshmallows0,80,078,5304
paste0,50,080,8310

Raw materials and seasonings

honey0,80,081,5329
sugar0,00,099,7398
milk sauce2,07,15,284

Dairy

milk3,23,64,864
kefir3,42,04,751
cream2,820,03,7205
sour cream2,820,03,2206
curdled milk2,92,54,153

Cheeses and cottage cheese

cottage cheese17,25,01,8121

Meat products

boiled beef25,816,80,0254
beef liver17,43,10,098
boiled beef tongue23,915,00,0231
boiled veal30,70,90,0131
rabbit21,08,00,0156

Bird

boiled chicken25,27,40,0170
turkey19,20,70,084

Eggs

chicken eggs12,710,90,7157

Oils and fats

butter0,582,50,8748
ghee0,299,00,0892

Non-alcoholic drinks

coffee with milk and sugar0,71,011,258
black tea with milk and sugar0,70,88,243

Juices and compotes

apricot juice0,90,19,038
carrot juice1,10,16,428
pumpkin juice0,00,09,038
* data is per 100 g of product

Classification of forms of erosive gastritis

  • Primary erosions (have no connection with previously identified pathology) and secondary (the result of progression, existing disease).
  • Malignant (appears in the presence of an oncological process) and benign (can be single or multiple).
  • Immature (young formations) and mature (have areas of tissue necrosis).

Based on the type and shape of formations on the mucous membrane, the following types are distinguished:

  • superficial (small, flat in shape and with a distinct edge);
  • full (raised above the surface of the mucosa);
  • polypoid (can have a diameter of up to one and a half centimeters, do not heal for a long time and constantly worsen).

Definition

Erosive gastritis is one of the types of inflammatory process of the gastric mucosa, in which superficial erosions are formed on it, which is accompanied by a violation of the secretory, endocrine, motor and evacuation functions of the organ. A gastroenterologist treats acute and chronic pathologies of the gastrointestinal tract. The Department of Gastroenterology at the Elena Malysheva Clinic in Izhevsk specializes in providing consultative, diagnostic and therapeutic care to patients with diseases of the digestive system, including erosive gastritis.

Symptoms of erosive gastritis

Patients suffering from erosive gastritis can present many different complaints, both specific and nonspecific:

  • nausea and rare episodes of vomiting
  • decreased appetite or complete absence of it
  • increased gas formation
  • heartburn
  • belching
  • pain in the epigastrium on an empty stomach or an hour and a half after eating
  • sudden weight loss
  • black stool due to bleeding
  • vomiting black-brown mass

Patients may also complain of increased drowsiness and fatigue, decreased tolerance to usual stress, the appearance of shortness of breath and tachycardia. Such symptoms may indicate the presence of hidden bleeding.

The pathology symptoms listed above indicate that the disease may have symptoms of gastric bleeding and the presence of mucosal defects.

Drug therapy

Drugs to improve gastric motor function A number of well-known experts have recommended a special method for the treatment of erosive gastritis, approved by WHO. The duration of this course of therapy is 14-15 days. Aggressive treatment is carried out with proton pump inhibitors, histamine receptor blockers and prokinetics. In the first stage, a triple regimen is used, consisting of one drug such as a proton pump inhibitor and two antibacterial substances: amoxicillin and clarithromycin.

If the first regimen does not radically change the situation, then a course of therapy is prescribed according to the second regimen, which includes a complex of 4 drugs: bismuth dicitrate, proton pump inhibitor, tetracycline and metronidazole. Antisecretory drugs and antacids reduce the activity of oxidized gastric juice, reduce total acidity and stop tissue erosion and erosion of the mucous membrane. The drugs Maalox, Almagel, Gelusil, Renicidin, Omeprazole, Gaviscon and some others have shown sufficient effectiveness in this direction. Digestion can be stabilized by using enzyme-type products: mezim, festal, panzinorm, etc.

For patients with a severe stage of the disease, intravenous administration of liquid antacids and blockers such as cimetidine, famotidine is provided; blood replacement drugs are used. Painful symptoms of a spasmodic nature are relieved with the help of antispasmodics: no-shpa, papaverine.

How is erosive gastritis diagnosed?

Erosive gastritis has symptoms similar to other gastrointestinal diseases. Therefore, to clarify the diagnosis, additional examination is prescribed. Most often, doctors recommend the following laboratory and instrumental studies:

  • a general blood test to determine the presence of anemia;
  • determination of occult blood in feces;
  • taking a section of the mucosa for cytology and histology;
  • PCR for diagnosing Helicobacter pylori infection;
  • fibrogastroscopy (FGDS) with a biopsy of the inflamed area;
  • radiography with contrast.

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Diagnostics

To determine the type of gastritis, the first step is endoscopy. Then blood and stool tests are mandatory. When diagnosis is difficult, X-ray examination is sometimes used.

Diagnosis of gastritis should only be carried out by a gastroenterologist. You cannot independently diagnose yourself or someone else, unless, of course, you have special education. Self-medication and self-diagnosis without medical education often leads to deaths.

Treatment of erosive gastritis

For the treatment of pathology, conservative tactics are usually chosen. Surgery is very rarely required.

The basis of successful treatment is diet and regular medication. Doctors usually prescribe a set of tablets to destroy Helicobacter pylori, relieve symptoms of the disease and stimulate healing of the mucous membrane.

Since Helicobacter pylori is an antibiotic-resistant microorganism, a combination of three or four drugs is chosen to destroy it. Treatment is not easy and takes quite a long time.

If there is bleeding, in addition to taking medications, the doctor may recommend thermo- or electrocoagulation of the mucous membrane, laser exposure, staples or endoscopic stitching.

During treatment of the pathology, the patient should adhere to a fractional diet plan. There should be at least 5 small meals a day. To reduce the likelihood of mechanical damage to the mucous membrane during healing, you should eat grated food or cooked with steam. It is also necessary to avoid eating too hot or cold foods, and avoid eating salty, fried, fatty and spicy foods. Sodas, strong coffee, tea and alcoholic drinks are not allowed.

Folk remedies

Traditional medicine can be a good help for drug therapy in restoring tissue and normalizing stomach functions, but if used correctly. It is best if folk remedies are agreed upon with your doctor. The following folk methods are widely used:

  1. Potato juice is taken 3 times a day before meals, and the dose is gradually increased from 25 ml to 100 ml, the course of treatment is 15 days.
  2. Sea buckthorn in the form of berries.
  3. Sea buckthorn oil is taken 3 times a day, 1 tbsp. l.
  4. Cabbage juice is taken 100 ml 3 times a day for 20-30 days.
  5. A mixture of flaxseeds with water (2 tablespoons per 0.5 liters of water), consumed 60-70 ml 3-4 times a day before meals for 15-25 days.
  6. Honey can be used separately or added to any composition.
  7. Sea buckthorn infusion: the berries are poured with boiling water (in a ratio of 1:5) and infused for 2 hours, honey is added to the cooled composition, and drunk in a volume of 0.8-1 liters during the day.

Erosive gastritis is a fairly dangerous disease and is one of the most severe types of gastritis. The effectiveness of treatment largely depends on the timely initiation of treatment. At the first signs, you should consult a doctor, since self-diagnosis is very difficult due to the similarity of symptoms with many other diseases.

Prevention

You can prevent the onset of the disease by observing personal hygiene measures, maintaining your diet, and giving up smoking and drinking alcoholic beverages. When working in hazardous industries, you should take precautions. When diagnosing diseases, carry out their timely treatment. You should also avoid stressful situations.

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Fully or partially limited products

The diet for erosive gastritis with high acidity involves excluding from the diet:

  • Raw vegetables (white cabbage, radishes, tomatoes, radishes, rutabaga, turnips, beans, onions, peas, sorrel, garlic), mushrooms and hard-skinned fruits as they contain coarse dietary fiber and irritate the gastric mucosa.
  • Products containing large quantities of connective tissue (skin, veins, cartilage) are difficult to digest.
  • Highly extractive meat/fish broths, borscht, cabbage soup, okroshka.
  • Animal/cooking fats, mayonnaise, margarine.
  • Fatty varieties of red meat, sea/river fish, duck, goose, smoked meats, canned meat/fish.
  • Coarse types of pasta and cereals (millet, pearl barley, corn, barley).
  • Hard-boiled/fried eggs are difficult to digest foods.
  • Pickled, pickled, salted vegetables, canned vegetables.
  • Mustard, horseradish, hot spices, tomato sauce, which irritate the gastric mucosa.
  • Butter/puff pastry, fresh white and rye bread.
  • Fermented milk products that have high acidity, as they stimulate the secretion of gastric juice.
  • Ice cream, sour fruits/berries, chocolate, carbonated drinks, alcohol.

Table of prohibited products

Proteins, gFats, gCarbohydrates, gCalories, kcal

Vegetables and greens

vegetables legumes9,11,627,0168
swede1,20,17,737
cabbage1,80,14,727
sauerkraut1,80,14,419
green onion1,30,04,619
bulb onions1,40,010,441
cucumbers0,80,12,815
canned cucumbers2,80,01,316
white radish1,40,04,121
turnip1,50,16,230
canned tomatoes1,10,13,520
horseradish3,20,410,556
spinach2,90,32,022
sorrel1,50,32,919

Mushrooms

mushrooms3,52,02,530

Cereals and porridges

corn grits8,31,275,0337
pearl barley9,31,173,7320
millet cereal11,53,369,3348
barley grits10,41,366,3324

Confectionery

candies4,319,867,5453

Ice cream

ice cream3,76,922,1189

Cakes

cake4,423,445,2407

Raw materials and seasonings

mustard5,76,422,0162
ginger1,80,815,880
ketchup1,81,022,293
mayonnaise2,467,03,9627
ground black pepper10,43,338,7251
chilli2,00,29,540

Dairy

sour cream2,820,03,2206

Meat products

pork16,021,60,0259
ham22,620,90,0279

Sausages

dry-cured sausage24,138,31,0455
sausages10,131,61,9332
sausages12,325,30,0277

Bird

smoked chicken27,58,20,0184
duck16,561,20,0346
smoked duck19,028,40,0337
goose16,133,30,0364

Fish and seafood

dried fish17,54,60,0139
smoked fish26,89,90,0196
canned fish17,52,00,088

Oils and fats

animal fat0,099,70,0897
cooking fat0,099,70,0897

Non-alcoholic drinks

bread kvass0,20,05,227
* data is per 100 g of product

Why are gastroprotectors needed?

To prevent complications such as peptic ulcers and malignant tumors, it is important to keep the structure of the mucous membrane intact. Gastroprotectors are used for these purposes:

  • adsorbent (creating an additional protective film);
  • astringents;
  • stimulating mucus formation;
  • antiseptic.
  • They should be taken when signs of gastritis appear. The doctor will determine which medications will fit into the selected treatment package. For prevention purposes, you do not need to drink them.

    The stomach contains natural gastroprotectors - prostaglandins. They stimulate the formation of mucus, improve microcirculation and protect cells from destruction. If they are not produced enough, the gastroenterologist may prescribe their synthetic analogues. If the patient is being treated with non-steroidal anti-inflammatory drugs, drugs with prostaglandins prevent ulcerative lesions of the mucous membrane.

    List of sources

    • Nikiforov P. A. et al. Diagnosis and follow-up of chronic erosions of the antrum of the stomach // Kremlin Medicine. Clinical Bulletin. – 2008. – No. 4. – P. 61-64.
    • Belova E.V. Characteristics of aggressive-protective factors in erosions of the gastroduodenal mucosa / EB Belova, Ya.M. Vakhrushev // Therapist. archive. 2002. - No. 2- P. 17-20.
    • Mosina, L. M. Some pathogenetic mechanisms of erosive and ulcerative lesions of the gastroduodenal zone / L. M. Mosina, L. V. Matveeva, E. A. Mitina // International Journal of Experimental Education. – 2011. – No. 7. – P. 40-42
    • Svinitsky, A. S. Stomach erosions: issues of pathogenesis, clinical picture, diagnosis and treatment / A. S. Svinitsky, G. A. Solovyova // Clinical medicine. – 2008. – T. 86, No. 9. – P. 18-24.
    • Maev I.V. Chronic gastric erosions: mechanisms of etiopathogenesis and new approaches to treatment // Clinical perspectives of gastroenterology and hepatology. - 2002. - No. 2. - P. 15-20.

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